Cellularized epithelial-fate lifecycle
Increment-4 mechanism — H→I→D + D→H Allee recovery (seed 17, 200 updates)
Increment 4
dead cells (H→I→D lesion, 200 updates)
0 → 14
organic D→H recovery events
3
direct H→D Allee deaths (rare, not a bug)
0
Epithelial-fate composition (population conserved every update)
Allee-driven transitions accrue organically (no hand-made geometry)
Mechanism validated, reproduction PENDING. The lifecycle composes: infection seeds I cells, infected-death converts them to D (n_D 0→14 over 200 updates, n_H 855→668, n_I 45→218, population conserved every step). D→H Allee recovery fires organically on local contact geometry (3 events here; a deterministic negative control confirms a dead cell surrounded only by dying neighbours never recovers). Direct H→D Allee death is genuinely rare at 0.3 mm (0 events) — a source-literal b_h/θ asymmetry plus this driver's per-update cadence, verified NOT a wiring bug (~0.19 expected successes over the run). Dominant death path is infection, not Allee death. No Fig 3B/5/7 target is claimed (deferred to Increment 9).